Skip to main content

Public Inquiry Needed

I have tabled this edm (see link)

That this House notes that in an email dated 24th October 2000, John Radford, Doncaster's then Director of Public Health, described the issue of research on babies by Dr David Southall at Doncaster Hospital in the late 1980s as `potentially a hot potato as to my recall the intervention resulted in increased deaths and didn't have proper consent'; expresses concern that the details of this research and its outcomes have been covered up by the health authorities; expresses particular concern that the research protocol specifically required that no action be taken to prevent cot death in the children selected until sufficient data had been collected; and calls for a public inquiry into this and other research managed by Dr Southall to identify why the checks and balances in the system failed

I obtained some information from Doncaster Hospital. Two pages of which are available on the internet here.

If you read those two documents one of which is an email sequence and the other of which is an extract from the research protocol you will see that they justify the claims in the EDM.

Comments

Popular posts from this blog

Its the long genes that stop working

People who read my blog will be aware that I have for some time argued that most (if not all) diseases of aging are caused by cells not being able to produce enough of the right proteins. What happens is that certain genes stop functioning because of a metabolic imbalance. I was, however, mystified as to why it was always particular genes that stopped working. Recently, however, there have been three papers produced: Aging is associated with a systemic length-associated transcriptome imbalance Age- or lifestyle-induced accumulation of genotoxicity is associated with a generalized shutdown of long gene transcription and Gene Size Matters: An Analysis of Gene Length in the Human Genome From these it is obvious to see that the genes that stop working are the longer ones. To me it is therefore obvious that if there is a shortage of nuclear Acetyl-CoA then it would mean that the probability of longer Genes being transcribed would be reduced to a greater extent than shorter ones.