Skip to main content

Results: Thursday 15th January 2009.

Forest of Dean DC, Newent Central
Con 306 (49.2; +6.5)
No Description 166 (26.7; -13.5)
Lab 96 (15.4; -1.7)
No Description 54 (8.7; +8.7)
Majority 140
Turnout 22%
Con hold
Percentage change is since May 2007

Haringey LBC, Seven Sisters
Lab 1032 (37.1; -9.3)
Con 968 (34.8; +7.0)
LD David Schmitz 581 (20.9; +8.1)
Green 166 (6.0; -7.1)
Ind 36 (1.3; +1.3)
Majority 64
Turnout 31.1%
Lab hold
Percentage change is since May 2006

Bishop's Stortford TC, Silver Leys
Con 653 (70.3)
LD Mat Harris 194 (20.9)
Lab 82 (8.8)
Majority 459
Turnout 22.7%
Con hold

Comments

Post a Comment

Popular posts from this blog

Its the long genes that stop working

People who read my blog will be aware that I have for some time argued that most (if not all) diseases of aging are caused by cells not being able to produce enough of the right proteins. What happens is that certain genes stop functioning because of a metabolic imbalance. I was, however, mystified as to why it was always particular genes that stopped working. Recently, however, there have been three papers produced: Aging is associated with a systemic length-associated transcriptome imbalance Age- or lifestyle-induced accumulation of genotoxicity is associated with a generalized shutdown of long gene transcription and Gene Size Matters: An Analysis of Gene Length in the Human Genome From these it is obvious to see that the genes that stop working are the longer ones. To me it is therefore obvious that if there is a shortage of nuclear Acetyl-CoA then it would mean that the probability of longer Genes being transcribed would be reduced to a greater extent than shorter ones.
1 comment
Read more