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Results: Thursday 8th January 2009.

Enfield LBC, Bush Hill Park
Con 1320 (63.4; +25.4)
Lab 413 (19.8; +6.7)
LD Paul Smith 129 (6.2; -3.0)
UKIP 123 (5.9; +0.9)
Green 97 (4.7; -5.5)
[Save Chase Farm (0.0; -24.4)]
Majority 907
Turnout 20.5%
Con hold
Percentage change is since May 2006.

Hull City UA, Drypool
LD Linda Chambers 1306 (52.3; -11.6)
Lab 891 (35.7; +7.9)
National Front 184 (7.4; +7.4)
Con 117 (4.7; -3.7)
Majority 415
Turnout 26.7%
LD hold (Andy Sloan was elected LD in 2006 but subsequently defected to Conservatives and later resigned.)
Percentage change is since May 2008.

Further result from 18th December 2008.

East Northamptonshire DC, Thrapston Lakes
Con 475 (63.0; +3.5)
Lab 168 (22.3; +22.3)
UKIP 111 (14.7; -25.8)
Majority 307
Turnout 22.4%
Con hold
Percentage change is since May 2007.

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Its the long genes that stop working

People who read my blog will be aware that I have for some time argued that most (if not all) diseases of aging are caused by cells not being able to produce enough of the right proteins. What happens is that certain genes stop functioning because of a metabolic imbalance. I was, however, mystified as to why it was always particular genes that stopped working. Recently, however, there have been three papers produced: Aging is associated with a systemic length-associated transcriptome imbalance Age- or lifestyle-induced accumulation of genotoxicity is associated with a generalized shutdown of long gene transcription and Gene Size Matters: An Analysis of Gene Length in the Human Genome From these it is obvious to see that the genes that stop working are the longer ones. To me it is therefore obvious that if there is a shortage of nuclear Acetyl-CoA then it would mean that the probability of longer Genes being transcribed would be reduced to a greater extent than shorter ones.