Skip to main content

Election Results 20th August 2009

Forest of Dean DC, Mitcheldean and Drybrook
LD Sue Henchley 638 (55.1; +36.2)
Ind 239 (20.6; -18.2)
Con 195 (16.8; -5.8)
Lab 86 (7.4; -12.1)
Majority 399
Turnout 32.4%
LD gain from Ind
Percentage change is since May 2007

Blackpool UA, Stanley
Con 648 (32.8; -28.2)
Lab 602 (30.5; +4.6)
LD Michael Hodkinson 332 (16.8; +3.6)
UKIP 203 (10.3; +10.3)
BNP 192 (9.7; +9.7)
Majority 46
Turnout 36.4%
Con hold
Percentage change is since May 2007

West Lindsey DC, Saxilby
Con 722 (60.6; +22.4)
LD Dean John Woolley 407 (34.2; -27.6)
UKIP 62 (5.2; +5.2)
Majority 315
Turnout 28.3%
Con gain from LD
Percentage change is since May 2008

Ashfield DC, Hucknall Central
LD Kevin Moore 463 (32.3; +18.8)
Lab 392 (27.3; +6.8)
Con 320 (22.3; -7.7)
UKIP 181 (12.6; +12.6)
Ind 79 (5.5; -30.5)
Majority 71
Turnout 26.16%
LD gain from Ind
Percentage change is since May 2007

Comments

Post a Comment

Popular posts from this blog

Its the long genes that stop working

People who read my blog will be aware that I have for some time argued that most (if not all) diseases of aging are caused by cells not being able to produce enough of the right proteins. What happens is that certain genes stop functioning because of a metabolic imbalance. I was, however, mystified as to why it was always particular genes that stopped working. Recently, however, there have been three papers produced: Aging is associated with a systemic length-associated transcriptome imbalance Age- or lifestyle-induced accumulation of genotoxicity is associated with a generalized shutdown of long gene transcription and Gene Size Matters: An Analysis of Gene Length in the Human Genome From these it is obvious to see that the genes that stop working are the longer ones. To me it is therefore obvious that if there is a shortage of nuclear Acetyl-CoA then it would mean that the probability of longer Genes being transcribed would be reduced to a greater extent than shorter ones.
1 comment
Read more