Skip to main content

Election Results 3rd September 2009

Medway UA, Luton & Wayfield
Con 1042 (36.9; +10.8)
Lab 1038 (36.7; -12.3)
LD Gary Allanach 223 (7.9; +7.9)
UKIP 200 (7.1; +7.1)
BNP 186 (6.6; +6.6)
Ind 87 (3.1; -21.9)
Green 51 (1.8; +1.8)
Majority 4
Turnout 29.78%
Con gain from Lab
Percentage change is since May 2007

Wiltshire UA, Southwick
Ind 385 (37.2; +37.2)
LD Gordon King 315 (30.5; -8.8)
Con 273 (26.4; -34.3)
UKIP 61 (5.9; +5.9)
Majority 70
Turnout 30.66%
Ind gain from Con
Percentage change is since June 2009

Plymouth City UA, Ham
Lab 1243 (44.0; +2.7)
Con 676 (23.9; -15.7)
UKIP 442 (15.6; +15.6)
BNP 82 (2.9; +2.9)
Ind 204 (7.2; +7.2)
LD Rebecca Trimnell 181 (6.4; -6.8)
[Green (0.0; -5.9)]
Majority 567
Turnout 29%
Lab hold
Percentage change is since May 2008

Stamford TC, St Georges
Ind 243 (42.4)
Con 135 (23.6)
LD Raymond Donald Lee 115 (20.1)
No Description 80 (14.0)
Majority 108
Turnout 17.7%
Ind hold

Party defending seat: Ind. Cause: Death.

Oswestry TC, Castle
Con 214 (61.3)
LD Brain L Willis 135 (38.7)
Majority 79
Turnout 15.46%
Con hold

Malvern Hills DC, Hallow
LD Dean Clarke 504 (61.6; +16.5)
Con 314 (38.4; -16.5)
Majority 190
Turnout not known
LD gain from Con
Percentage change is since May 2007

Melksham TC, Melksham Spa
LD Peter Dauncey 732 (58.8)
Con 432 (34.7)
BNP 81 (6.5)
Majority 300
Turnout 26%
LD gain from Con
Party defending seat: Con. Cause: Resignation.

Chippenham TC, Chippenham Park
LD Lorraine Roberts-Rance 427 (66.4)
Con 216 (33.6)
Majority 211
Turnout 30.03%
LD hold
Party defending seat: LD. Cause: Resignation.

Wootton Bassett TC, North
LD Stephen Richard Walls 423 (56.0)
Con 333 (44.0)
Majority 90
Turnout 22%
LD gain from Con
Part defending seat: Con. Cause: Disqualification.

Calne TC, Quemerford
Con 299 (53.9)
LD Sally Ann Chandler 256 (46.1)
Majority 43
Turnout 29.8%
Con hold
Party defending seat: Con. Cause: Resignation.

Comments

Popular posts from this blog

Its the long genes that stop working

People who read my blog will be aware that I have for some time argued that most (if not all) diseases of aging are caused by cells not being able to produce enough of the right proteins. What happens is that certain genes stop functioning because of a metabolic imbalance. I was, however, mystified as to why it was always particular genes that stopped working. Recently, however, there have been three papers produced: Aging is associated with a systemic length-associated transcriptome imbalance Age- or lifestyle-induced accumulation of genotoxicity is associated with a generalized shutdown of long gene transcription and Gene Size Matters: An Analysis of Gene Length in the Human Genome From these it is obvious to see that the genes that stop working are the longer ones. To me it is therefore obvious that if there is a shortage of nuclear Acetyl-CoA then it would mean that the probability of longer Genes being transcribed would be reduced to a greater extent than shorter ones.